Photo
Language
English
Title and Department
Associate Professor of Medicine
Department of Medicine, Division of Infectious Diseases
Professional bio
Carlos Serezani, PhD, is an Associate Professor of Medicine in the Division of Infectious Diseases within the Department of Medicine at Vanderbilt University Medical Center.
He completed his PhD at the University of Sao Paulo and the University of Michigan, and completed his postdoctoral studies at University of Michigan. Before joining VUMC, he was an assistant professor at Indiana University School of Medicine and has published more than 50 papers in reputed journals.
The NIH and other foundations have funded Dr. Serezani’s laboratory for almost 10 years. The Serezani lab aims to develop therapeutic strategies to control systemic (sepsis) and localized infections (skin and lung) in healthy individuals, individuals with immune deficiencies, and those suffering from chronic inflammatory diseases, such as diabetes. They have concentrated much of their efforts on understanding the role of the lipid mediators leukotriene B4 and prostaglandin E2 and their actions on microRNAs and phosphatases to modulate immune cells involved in the control of microbial infection in these different contexts.
He completed his PhD at the University of Sao Paulo and the University of Michigan, and completed his postdoctoral studies at University of Michigan. Before joining VUMC, he was an assistant professor at Indiana University School of Medicine and has published more than 50 papers in reputed journals.
The NIH and other foundations have funded Dr. Serezani’s laboratory for almost 10 years. The Serezani lab aims to develop therapeutic strategies to control systemic (sepsis) and localized infections (skin and lung) in healthy individuals, individuals with immune deficiencies, and those suffering from chronic inflammatory diseases, such as diabetes. They have concentrated much of their efforts on understanding the role of the lipid mediators leukotriene B4 and prostaglandin E2 and their actions on microRNAs and phosphatases to modulate immune cells involved in the control of microbial infection in these different contexts.