Title and Department
Osaka University School of Medicine
Professional bio
Hideaki worked on trafficking of cardiac potassium channels. His studies identified a key structural motif in the C-terminus of the KCNQ1 channel that is responsible for normal processing of the channel to the cell surface. Mutations in this motif result in failure of KCNQ1 channels to process normally to the cell surface, and thus are a common mechanism for the long QT syndrome, type 1. Hideaki is currently at Keio University School of Medicine in Tokyo, Japan.